DIFFERENTIAL EFFECTS OF H. PYLORI ERADICATION ON OXIDATIVE DNA DAMAGE AT THE GASTROESOPHAGEAL JUNCTION AND AT THE GASTRIC ANTRUM
This is another article in my “lone crusade to prove that H. pylori is merely an opportunistic infection and the real problem is the environment that allowed H. pylori to grow and prosper.”
However, this article goes even further then my lone crusade. What if, in some cases, having H. pylori present is actually PROTECTIVE? Now wouldn’t that be a novel concept. I’m not quite to this extreme yet, but this article found an increase in oxidative DNA damage when H. pylori was eradicated. This would lead to an increase in the risk of cancer at the GE junction.
Stay tuned… Differential Effects of Helicobacter pylori Eradication on Oxidative DNA Damage at the Gastroesophageal Junction and at the G..