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Almost a 50% response with taking 300 mg CoQ10 per day.  Not too shabby.  This is yet another notch in the "mitochondrial dysfunction leads to neurodegenerative disorders" belt.  The research is no longer questioning the association between altered brain behavior/pathology and oxidative stress--the research is now moving onto the deeper, more practical questions.

 

 

***Lifestyle factors and the development of bone mass and bone strength in young women***
Geez, I hate to see articles that support what I've been saying for years...  I would have to say, that, of all the myths I bring to light in the community and my office, few receive more resistance than the idea that dairy is actually, at best, neutral, and at worst, harmful.  This article, while small, did not find that calcium intake had any impact on bone development (milk for bone health, anyone?  Did see THIS one in the mainstream press, did you?).  Number one beneficial item?  Exercise.

 

 

Every normal cell has the ability to undergo programmed cell death called apoptosis.  Too much apoptosis plays a role in neuron death in Alzheimer's, Parkinson's, ALS and seizures.  This review reminds us how incredibly important zinc is in keeping this in check.  Zinc deficiency in mice leads to a 300% increase in cell death, including a nasty form called autophagy, where the cell feeds off its own organelles to survive.  This really, once again, supports the use of a good quality multivitamin as an absolute essential component of good health.

 

While this should not come as a surprise, anything I can put out on pancreatic cancer prevention is important.  This is still such a devastating cancer without a long list of  identified modifyable risk factors like other cancers.  Aspirin is also known to increase risk of pancreatic cancer.

 

 

Here's some background...the body metabolizes estrogens down several pathways.  The "2" pathway is considered protective for certain types of cancer while the "16" pathway is damaging.  It is known that equols, compounds produced by bacterial conversion of soy isoflavones in the gut, shift the metabolization of estrogen to the 2 pathway (so does indole-3-carbonol from cruciferous veggies).  So, the idea in this study was to give probiotics with soy to see how much of an effect on equol levels was seen.  There seems to be a subset of women that respond positively to this approach, but the researchers were not able to identify specifics.  I do have a question for the more research-minded readers out there...This study used a 2 week washout period.  How can you get a washout period using probiotics?  All things being healthy--the probiotics should maintain in the GI tract.  I wonder how much this influenced the results?

 

 

Can someone explain to me why, in study after study after study, the parents who chose not to follow the full vaccination schedule are the most educated and more affluent?  Just a rhetorical question, of course...

 

 

***Assessment of the efficacy and effectiveness of influenza vaccines in healthy children: systematic review***

To quote: "no studies assessed reductions in mortality, serious complications."  Well, isn't that just wonderful considering how much of a push there is to get every child vaccinated with the flu vaccine. Compare this with a recent review of the flu vaccine in the elderly used over the past 40 years or so with no reduction in mortality in those recieving the vaccine.  And remember this next flu season--the push will be on again to get vaccinated and the scare tactics will be employed.  We'll forget these pesky little studies and line up like lemmings...

 

 

Wow.  What an insightful approach.  Basically, visceral (bad) and subcutaneous fat have a different response in the level of release of hormones after a glucose load.  The hormones derived from adipose tissue (resistin, leptin, adinopectin, PAI-1) generally contribute to insulin resistance and obesity.  More is released by visceral fat after feeding.  So the authors have planted a seed--might checking blood levels in non-fasting states give us more insight into the balance of visceral vs subcutaneous fat?  Interesting, huh?

 

 

***Increased Prevalence of Celiac Disease and Need for Routine Screening Among Patients With Osteoporosis***

Remember that celiac disease is an allergy to the gluten portion in grains, principally wheat.  This study found a direct correlation with the severity of celiac disease serum markers and the severity of osteoporosis.  We already know that inflammation is a player in osteoporosis and that celiac disease upregulates the immune system, so I guess the results of this study shouldn't be a surprise.  The author suggests that the prevalance of patients with osteoporosis that have celiac disease is high enough to warrant routine screening.